Oligodendrocyte damage and subsequent axonal demyelination is a hallmark of this disease.
少突胶质细胞损害,继而轴突脱髓鞘为本病特征.
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Conclusion EAE is characterised by perivascular inflammatory cell infiltration and demyelination in white matter.
结论EAE的病理改变主要为血管周围炎性浸润及白质脱髓鞘.
Significant endoneurial edema, inflammation reaction and demyelination can be seen in the DRGs and NRs.
自体髓核移植可致脊神经节、神经根明显的水肿 、 炎性反应、脱髓鞘等病理改变.
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